The unprotected handling of UV radiation promotes premature skin aging, pigmentation disorders and the development of skin cancer.

Disease of the sebaceous gland apparatus and hair follicles, which initially develop non-inflammatory comedons, in the further course inflammatory papules and pustules. With severe course, scarring is possible.

Melanocytes – the birthplace of melanin: pigment cells of the skin, located in the basal cell layer of the epidermis and the hair follicles, synthesize melanin and release it in the form of so-called melanosomes to the surrounding keratinocytes. As a result, stimulation causes an increased brown coloration of the skin.

Endogenous / chronological skin aging: internal influences, physiological skin aging e.B. predisposition, hormone balance, wrinkle formation, loss of contours. Exogenous / premature skin aging: external influences e.B. hyperpigmentation, damage to collagen and elastin.

Braid-like expansion of the dilated capillaries of the skin. Increased occurrence over the cheekbones, nostrils, nasal entrance and cheeks. Sensitive skin prone to allergies reacts quickly with redness, tenses, itches and flakes.

With dry skin, the skin’s natural fat and moisture regulation is disrupted. The skin becomes rough and brittle, tenses and, in extreme cases, forms cracks and dehydration eczema.

Endogenous / chronological skin aging: internal influences, physiological skin aging e.B. predisposition, hormone balance, wrinkle formation, loss of contours. Exogenous / premature skin aging: external influences e.B. hyperpigmentation, damage to collagen and elastin.