Skincare
More than Beauty
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SERIES BY SKIN SITUATION
UV-Protect
The unprotected handling of UV radiation promotes premature skin aging, pigmentation disorders and the development of skin cancer.
UV-Protect
UV-Protect
The unprotected handling of UV radiation promotes premature skin aging, pigmentation disorders and the development of skin cancer.
Oily, impure acne-prone skin
Disease of the sebaceous gland apparatus and hair follicles, which initially develop non-inflammatory comedons, in the further course inflammatory papules and pustules. With severe course, scarring is possible.
Oily, impure acne-prone skin
Oily, impure acne-prone skin
Disease of the sebaceous gland apparatus and hair follicles, which initially develop non-inflammatory comedons, in the further course inflammatory papules and pustules. With severe course, scarring is possible.
Hyperpigmented and sun-damaged skin
Melanocytes – the birthplace of melanin: pigment cells of the skin, located in the basal cell layer of the epidermis and the hair follicles, synthesize melanin and release it in the form of so-called melanosomes to the surrounding keratinocytes. As a result, stimulation causes an increased brown coloration of the skin.
Hyperpigmented and sun-damaged skin
Hyperpigmented and sun-damaged skin
Melanocytes – the birthplace of melanin: pigment cells of the skin, located in the basal cell layer of the epidermis and the hair follicles, synthesize melanin and release it in the form of so-called melanosomes to the surrounding keratinocytes. As a result, stimulation causes an increased brown coloration of the skin.
Anti-aging from 25+
Endogenous / chronological skin aging: internal influences, physiological skin aging e.B. predisposition, hormone balance, wrinkle formation, loss of contours. Exogenous / premature skin aging: external influences e.B. hyperpigmentation, damage to collagen and elastin.
Anti-aging from 25+
Anti-aging from 25+
Endogenous / chronological skin aging: internal influences, physiological skin aging e.B. predisposition, hormone balance, wrinkle formation, loss of contours. Exogenous / premature skin aging: external influences e.B. hyperpigmentation, damage to collagen and elastin.
Couperose and sensitive skin prone to allergies
Braid-like expansion of the dilated capillaries of the skin. Increased occurrence over the cheekbones, nostrils, nasal entrance and cheeks. Sensitive skin prone to allergies reacts quickly with redness, tenses, itches and flakes.
Couperose and sensitive skin prone to allergies
Couperose and sensitive skin prone to allergies
Braid-like expansion of the dilated capillaries of the skin. Increased occurrence over the cheekbones, nostrils, nasal entrance and cheeks. Sensitive skin prone to allergies reacts quickly with redness, tenses, itches and flakes.
Dry, dehydrated skin
With dry skin, the skin’s natural fat and moisture regulation is disrupted. The skin becomes rough and brittle, tenses and, in extreme cases, forms cracks and dehydration eczema.
Dry, dehydrated skin
Dry, dehydrated skin
With dry skin, the skin’s natural fat and moisture regulation is disrupted. The skin becomes rough and brittle, tenses and, in extreme cases, forms cracks and dehydration eczema.
Anti-aging from 45+
Endogenous / chronological skin aging: internal influences, physiological skin aging e.B. predisposition, hormone balance, wrinkle formation, loss of contours. Exogenous / premature skin aging: external influences e.B. hyperpigmentation, damage to collagen and elastin.
Anti-aging from 45+
Anti-aging from 45+
Endogenous / chronological skin aging: internal influences, physiological skin aging e.B. predisposition, hormone balance, wrinkle formation, loss of contours. Exogenous / premature skin aging: external influences e.B. hyperpigmentation, damage to collagen and elastin.
